KD TRIPATHI BOOK PDF
, KD Tripathi. Managing Editor: M. This book has been published in good faith that the material provided by author is original. Every effort is made to. (tripathi) essentials of medical pharmacology KD TRIPATHI MD. This, the 6th edition ot the Textbook of Medical Parasitology comes after 18 years By. I am not sure if this is what you wanted, ppti.info?sa= But if you have tried searching this in google, it could have took less.
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KD Tripathi is one of the books most widely considered in Pharmacology. Below are the links are given to KD Tripathi PDF format for. Pharmacology Kd Tripathi 7th Edition [PDF] [EPUB] Esmolol (trade name And Jelly Book 3Practical Telecommunications And Wireless. KD TRIPATHI MD. Ex-Director-Professor . diseases/conditions. By a unique synthesis of pharmacology with clinical medicine, the book is designed to be useful.
Rate of tubular secretion p.
More than the glomerular filtration rate B. Equal to the glomerular filtration rate C. Less than the glomerular filtration rate D. Equal to the rate of urine formation p. Plasma protein level is low B.
Drug metabolizing enzymes are immature C. Glomerular filtration rate is low D. Tubular transport mechanisms are not well p. Bioavailability B. Volume of distribution C. Clearance D. Plasma half life p. A constant amount of the drug will be eliminated per unit time B. Its clearance value will remain constant C. Its elimination half life will increase with dose D. It will be completely eliminated from the body in p.
Volume of distribution B. Lipid solubility D. Total body clearance p. Renal clearance B. Plasma half life C. Volume of distribution p. Elimination rate constant 2. No sensitive methods for measuring blood levels of diuretics are available B.
KD Tripathi 7th Edition Essentials Of Medical Pharmacology
It is easier to measure the effect of these drugs C. Response to diuretics is not related to their blood levels p. Diuretics need activation in the body 2. Antihypertensive drugs B. Levodopa C. Lithium carbonate p. MAO inhibitors 2.
An antiarthritic with a plasma half life of 24 hr B. A sleep inducing hypnotic with a plasma half life of 2 hours C. An antihypertensive with a plasma half life of 3 hours D.
An analgesic with a plasma half life of 6 hours used for relief of casual headache p. Takes about one week to develop B.
Results in increased affinity of the enzyme for the substrate C. It is irreversible D.
Can be used to treat acute drug poisonings p. Atropine B. Allopurinol C. Levodopa p. Metoclopramide 3. Acetazolamide B. Disulfiram C. Physostigmine p. Theophylline 2. All drugs act through specific receptors B. All drug receptors are located on the surface of the target cells C. Agonists induce a conformational change in the receptor D.
Partial agonists have low affinity for the receptor p. Structural specificity B. High potency C. Competitive antagonism p. Dependence of action on lipophilicity 3. Maximal response occurs only when all receptors are occupied by the drug B.
Drugs exert an all or none action on a receptor C. Receptor and drugs acting on it have rigid complementary lock and key structural features D. Properties of affinity and intrinsic activity are independently variable p.
High affinity but low intrinsic activity B. Low affinity but high intrinsic activity C. No affinity and low intrinsic activity D.
High affinity but no intrinsic activity p. Affinity but no intrinsic activity B. Phenylephrine B. Desmopressin C. Azelastine D. Beclomethasone dipropionate p. Is more painful B.
Produces faster response C. Is unsuitable for depot preparations D. Carries greater risk of anaphylactic reaction p. Intra arterial injection B. Intrathecal injection C.
Intravenous injection D. Intramuscular injection p. Weakly basic drugs B. Weakly acidic drugs C. Strong electrolytes D. Nonpolar drugs p. Passive diffusion B. Facilitated diffusion C. Active transport p. Pinocytosis 2. Is dependent upon metabolic activity of the cell B.
Is competitively inhibited by chemically related drugs C. Is affected by extent of ionization of drug molecules D. Exhibits saturation kinetics p. Morphine sulfate B. Diclofenac sodium C. Hyoscine hydrobromide D. Quinine dihydrochloride p. Atropine sulfate B. Chloroquine phosphate C.
Ephedrine hydrochloride p. Phenytoin sodium 2. Blood flow through the capillary B. Lipid solubility of the drug C. It is specific B. It is pH dependent C. It is saturable D. It requires metabolic energy p.
Complexing with the other drug in the intestinal lumen B. Altering gut motility C. Altering gut flora D. Damaging gut mucosa p. Percentage of administered dose that reaches systemic circulation in the unchanged form B. Ratio of oral to parenteral dose C. Ratio of orally administered drug to that excreted in the faeces D.
Ratio of drug excreted unchanged in urine to that excreted as metabolites p. Is freely water soluble B. Is completely absorbed C. Is incompletely absorbed D. Undergoes little first-pass metabolism p. Molecular weight of the drug B. Site of application C. Lipid solubility of the drug D. Nature of the base used in the formulation p. High lipid solubility B. Low ionisation at physiological pH values C.
High plasma protein binding D. High tissue binding p.
Highly lipid soluble drugs B. Poorly lipid soluble drugs C. Depot preparations D. Highly plasma protein bound drugs 2.
On intravenous injection it produces general anaesthesia for 10 min. Which process is responsible for termination of its action: A. Metabolism in liver B. Plasma protein binding C. Excretion by kidney D.
Redistribution p. P-glycoprotein efflux carriers in brain capillary cells B. Tight junctions between endothelial cells of brain capillaries C. Enzymes present in brain capillary walls D. All of the above p. It is constituted by tight junctions between the endothelial cells of brain capillaries and the glial tissue B.
It allows passage of lipid soluble drugs into the brain C.
KD Tripathi 7th Edition Essentials Of Medical Pharmacology
It limits entry of highly ionized drugs into the brain D. It regulates passage of substances from brain into blood p. Are excreted faster in alkaline urine C. Are highly ionized in the gastric juice D. Do not cross blood-brain barrier p. Increases volume of distribution of the drug B.
Facilitates glomerular filtration of the drug C. Notify me of new posts via email. Search for: Share this: WhatsApp Facebook Email. May 19, at July 1, at Plz send on Anurag19tripathi gmail.
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September 10, at 3: September 13, at Equal to the rate of urine formation p. July 16, at 5: Tubular transport mechanisms are not well p. High potency C. Sublingual B. Transdermal C. Nature of the base used in the formulation p.
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